PXD075372 is an
original dataset announced via ProteomeXchange.
Dataset Summary
| Title | Senescence inhibition by rapamycin mitigates radiation-induced atherosclerosis characteristics in human coronary endothelial cells |
| Description | HCAEC from 12 independent donors were irradiated with either 0 or 10 Gy X-ray, and received either 0 or 10 nM rapamycin treatment for 5 days, with cells harvested on day 7. |
| HostingRepository | PRIDE |
| AnnounceDate | 2026-06-24 |
| AnnouncementXML | Submission_2026-06-24_01:40:37.249.xml |
| DigitalObjectIdentifier | |
| ReviewLevel | Peer-reviewed dataset |
| DatasetOrigin | Original dataset |
| RepositorySupport | Unsupported dataset by repository |
| PrimarySubmitter | Christopher Whiteman |
| SpeciesList | scientific name: Homo sapiens (Human); NCBI TaxID: NEWT:9606; |
| ModificationList | No PTMs are included in the dataset |
| Instrument | Q Exactive |
Dataset History
| Revision | Datetime | Status | ChangeLog Entry |
| 0 | 2026-03-09 00:06:43 | ID requested | |
| ⏵ 1 | 2026-06-24 01:40:37 | announced | |
Publication List
| Dataset with its publication pending |
Keyword List
Contact List
| Christopher Whiteman |
| contact affiliation | UKHSA |
| contact email | christopher.whiteman@ukhsa.gov.uk |
| lab head | |
| Christopher Whiteman |
| contact affiliation | UK Health Security Agency |
| contact email | christopher.whiteman@ukhsa.gov.uk |
| dataset submitter | |
Full Dataset Link List
Dataset FTP location
NOTE: Most web browsers have now discontinued native support for FTP access within the browser window. But you can usually install another FTP app (we recommend FileZilla) and configure your browser to launch the external application when you click on this FTP link. Or otherwise, launch an app that supports FTP (like FileZilla) and use this address: ftp://ftp.pride.ebi.ac.uk/pride/data/archive/2026/06/PXD075372 |
| PRIDE project URI |
Repository Record List
[ + ]
[ - ]
- PRIDE
- PXD075372
- Label: PRIDE project
- Name: Senescence inhibition by rapamycin mitigates radiation-induced atherosclerosis characteristics in human coronary endothelial cells