PXD015992-1

PXD015992 is an original dataset announced via ProteomeXchange.

Title	Influenza virus infection leads to global cardiac proteome remodeling during convalescence
Description	Influenza virus infection leads to global cardiac proteome remodeling during convalescence MTD project_description "Influenza virus infections lead to more than 500,000 hospitalizations in the U.S. every year. Patients with cardiovascular diseases have been shown to be at high risk of influenza mediated cardiac complications. Importantly, recent reports have provided clinical data supporting a direct link between laboratory-confirmed influenza virus infection and adverse cardiac events. However, the molecular mechanisms of how influenza virus infection induces detrimental cardiac changes, even after resolution of the pulmonary infection, is completely unknown.
HostingRepository	PRIDE
AnnounceDate	2022-05-25
AnnouncementXML	Submission_2022-05-25_02:25:33.512.xml
DigitalObjectIdentifier
ReviewLevel	Peer-reviewed dataset
DatasetOrigin	Original dataset
RepositorySupport	Unsupported dataset by repository
PrimarySubmitter	Yanbao Yu
SpeciesList	scientific name: Mus musculus (Mouse); NCBI TaxID: 10090;
ModificationList	phosphorylated residue
Instrument	Q Exactive

Revision	Datetime	Status	ChangeLog Entry
0	2019-10-24 00:52:23	ID requested
⏵ 1	2022-05-25 02:25:34	announced
2	2024-10-22 05:36:45	announced	2024-10-22: Updated project metadata.

Lin YH, Platt MP, Gilley RP, Brown D, Dube PH, Yu Y, Gonzalez-Juarbe N, Influenza Causes MLKL-Driven Cardiac Proteome Remodeling During Convalescence. Circ Res, 128(5):570-584(2021) [pubmed]

submitter keyword: Influenza virus

proteome

remodeling

convalescence

heart

Yanbao Yu
contact affiliation	J. Craig Venter Institute, Rockville, MD 20850
contact email	yayu@jcvi.org
lab head
Yanbao Yu
contact affiliation	J. Craig Venter Institute
contact email	yayu@jcvi.org
dataset submitter

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PRIDE project URI

• PRIDE
  1. PXD015992
     1. Label: PRIDE project
     2. Name: Influenza virus infection leads to global cardiac proteome remodeling during convalescence