PXD056172 is an
original dataset announced via ProteomeXchange.
Dataset Summary
Title | Targeting KPNB1 suppresses DNA damage repair in AML cells by inhibiting HMGB2 nuclear translocation |
Description | To gain insight into the potential cargo proteins mediated by KPNB1, the nuclear fraction of DMSO- and IPZ-treated NB-4 cells were isolated and then subjected to mass spectrometry analysis. |
HostingRepository | PRIDE |
AnnounceDate | 2025-01-03 |
AnnouncementXML | Submission_2025-01-02_19:43:05.166.xml |
DigitalObjectIdentifier | |
ReviewLevel | Peer-reviewed dataset |
DatasetOrigin | Original dataset |
RepositorySupport | Unsupported dataset by repository |
PrimarySubmitter | Zhe Chen |
SpeciesList | scientific name: Homo sapiens (Human); NCBI TaxID: 9606; |
ModificationList | No PTMs are included in the dataset |
Instrument | time-of-flight |
Dataset History
Revision | Datetime | Status | ChangeLog Entry |
0 | 2024-09-24 06:21:53 | ID requested | |
⏵ 1 | 2025-01-02 19:43:05 | announced | |
Publication List
Dataset with its publication pending |
Keyword List
submitter keyword: KPNB1,NB-4 |
Contact List
Zhe Chen |
contact affiliation | Targeting KPNB1 suppresses DNA damage repair in AML cells by inhibiting HMGB2 nuclear translocation |
contact email | chenzhe925@cqmu.edu.cn |
lab head | |
Zhe Chen |
contact affiliation | Chongqing Medical University |
contact email | chenzhe925@cqmu.edu.cn |
dataset submitter | |
Full Dataset Link List
Dataset FTP location
NOTE: Most web browsers have now discontinued native support for FTP access within the browser window. But you can usually install another FTP app (we recommend FileZilla) and configure your browser to launch the external application when you click on this FTP link. Or otherwise, launch an app that supports FTP (like FileZilla) and use this address: ftp://ftp.pride.ebi.ac.uk/pride/data/archive/2025/01/PXD056172 |
PRIDE project URI |
Repository Record List
[ + ]
[ - ]
- PRIDE
- PXD056172
- Label: PRIDE project
- Name: Targeting KPNB1 suppresses DNA damage repair in AML cells by inhibiting HMGB2 nuclear translocation