PXD052176 is an
original dataset announced via ProteomeXchange.
Dataset Summary
Title | Targeting MGAT4A-mediated N-glycosylation as a therapeutic strategy to inhibit glioblastoma stem cell invasion |
Description | Proteomic glycosylation mapping via mass spectrometry and co-immunoprecipitation assays elucidates the MGAT4A interaction with EGFR, catalyzing N-glycosylation at the EGFR N604 site. |
HostingRepository | PRIDE |
AnnounceDate | 2025-01-22 |
AnnouncementXML | Submission_2025-01-22_10:31:01.383.xml |
DigitalObjectIdentifier | |
ReviewLevel | Peer-reviewed dataset |
DatasetOrigin | Original dataset |
RepositorySupport | Unsupported dataset by repository |
PrimarySubmitter | Jinlong Yin |
SpeciesList | scientific name: Homo sapiens (Human); NCBI TaxID: 9606; |
ModificationList | N-glycosylated residue |
Instrument | Q Exactive HF |
Dataset History
Revision | Datetime | Status | ChangeLog Entry |
0 | 2024-05-10 08:01:18 | ID requested | |
1 | 2025-01-17 18:17:13 | announced | |
⏵ 2 | 2025-01-22 10:31:01 | announced | 2025-01-22: Updated project metadata. |
Publication List
Dataset with its publication pending |
Keyword List
submitter keyword: Human, Cell, N-glycoproteomics, Label-free |
Contact List
Jinlong Yin |
contact affiliation | Henan University |
contact email | jlyin@henu.edu.cn |
lab head | |
Jinlong Yin |
contact affiliation | Henan University |
contact email | jlyin@henu.edu.cn |
dataset submitter | |
Full Dataset Link List
Dataset FTP location
NOTE: Most web browsers have now discontinued native support for FTP access within the browser window. But you can usually install another FTP app (we recommend FileZilla) and configure your browser to launch the external application when you click on this FTP link. Or otherwise, launch an app that supports FTP (like FileZilla) and use this address: ftp://ftp.pride.ebi.ac.uk/pride/data/archive/2025/01/PXD052176 |
PRIDE project URI |
Repository Record List
[ + ]
[ - ]
- PRIDE
- PXD052176
- Label: PRIDE project
- Name: Targeting MGAT4A-mediated N-glycosylation as a therapeutic strategy to inhibit glioblastoma stem cell invasion