PXD018742 is an
original dataset announced via ProteomeXchange.
Dataset Summary
| Title | MCU activity compensates for Complex I dysfunction |
| Description | Co-immunoprecipitation experiments were performed in HEK293T cells in the absence and presence of Flag-tagged MCU and binding proteins were identified by tandem mass spectrometry. |
| HostingRepository | PRIDE |
| AnnounceDate | 2024-10-22 |
| AnnouncementXML | Submission_2024-10-22_05:35:21.380.xml |
| DigitalObjectIdentifier | |
| ReviewLevel | Peer-reviewed dataset |
| DatasetOrigin | Original dataset |
| RepositorySupport | Unsupported dataset by repository |
| PrimarySubmitter | Ryan Bia |
| SpeciesList | scientific name: Homo sapiens (Human); NCBI TaxID: 9606; |
| ModificationList | No PTMs are included in the dataset |
| Instrument | LTQ Orbitrap Velos |
Dataset History
| Revision | Datetime | Status | ChangeLog Entry |
| 0 | 2020-04-22 15:22:46 | ID requested | |
| 1 | 2022-03-10 14:42:24 | announced | |
| ⏵ 2 | 2024-10-22 05:35:21 | announced | 2024-10-22: Updated project metadata. |
Publication List
| Dataset with its publication pending |
Keyword List
| submitter keyword: Mitochondria, Label-Free, Heart,Co-IP, LC-MS/MS |
Contact List
| Sarah Franklin |
| contact affiliation | CVRTI, University of Utah (lab head) |
| contact email | sarah.franklin@utah.edu |
| lab head | |
| Ryan Bia |
| contact affiliation | University of Utah |
| contact email | ryan.bia@utah.edu |
| dataset submitter | |
Full Dataset Link List
Dataset FTP location
NOTE: Most web browsers have now discontinued native support for FTP access within the browser window. But you can usually install another FTP app (we recommend FileZilla) and configure your browser to launch the external application when you click on this FTP link. Or otherwise, launch an app that supports FTP (like FileZilla) and use this address: ftp://ftp.pride.ebi.ac.uk/pride/data/archive/2022/03/PXD018742 |
| PRIDE project URI |
Repository Record List
[ + ]
[ - ]
- PRIDE
- PXD018742
- Label: PRIDE project
- Name: MCU activity compensates for Complex I dysfunction